G1 stage

Immunocytochemical staining results of rat CD molecules exhibited positive expression of CD44 and CD105 and negative expression of CD34 and CD45.Flow cytometry cell cycle analysis showed G0/ G1 stage of rat BMSCs accounting more than 90 percent.

免疫细胞化学染色法检测大鼠CD分子表达结果为CD44及CD105的阳性表达及CD34及CD45阴性表达，流式细胞周期分析显示G0/G1期细胞占90.18%。

Cell cycle alteration of Raji cells occurred in 131I-rituximab group, and the majority of cells were arrested at G1/ G2 stage.

131I-rituximab组Raji细胞周期发生变化，细胞大部分被阻滞于G1／G2期。

Androgen withdrawal might drive LNCaP cell into G1 stage.

激素撤退可引起LNCaP细胞G1期阻滞。

Conclusion: The expression of cyclinE and activity of cyclinE/ Cdk2 kinase are increased obviously in G0/ G1 stage cells infected with HCMV, which may induce the cell cycle to overpass G1/ S restriction point and make the cell cycle arrested in later G1 stage.

结论HCMV感染同步化于G0/G1期的细胞后，诱导CyclinE蛋白明显升高，激活CyclinE/Cdk2激酶，使细胞周期越过G1/S限制点，将细胞周期阻止于晚G1期。

Conclusion Lymphocyte could be arrested at G1/ S cycle stage and the growth of cell cycle could be suppressed by sulfur mustard. The mechanism might be carried out through sulfur mustard-induced apoptosis in lymphocyte.

结论硫芥可抑制体外培养的淋巴细胞由G1期进入S期，抑制体外培养的淋巴细胞增殖，通过诱导淋巴细胞凋亡实现作用机制。

Cell cycles were blocked in G1 stage. Conclusion Overexpression of PTEN will induce G1 arrest in rar renal fibroblast cells, which associated with the nuclear location of PTEN.

结论PTEN过度表达将引起肾脏成纤维细胞周期G1期阻滞，这一过程与PTEN在细胞核定位有关。

In the distributions figure of cell cycle, G1 stage cells apparently reduced.

细胞周期分布图中，G1细胞明显减少。

In current opinion, TGF-β 1 suppresses proto-oncogene such as c-myc to inhibit cell growth cycle of G1 stage to S stage.

目前认为，在肿瘤早期TGF-β1抑制原癌基因c-myc等，阻止细胞生长周期从G1期进入S期；

Zinc can promote osteoblasts transformation form G0/ G1-stage to S-stage and accelerate cells to enter DNA synthesis period.

锌能够促进成骨细胞从G0/G1期向S期转化，加速细胞进入DNA合成期，从而促进DNA合成及细胞增殖。

The cell cycle was blocked in G1 and G2 stage by SAL.

使细胞周期阻滞于G1、G2期；

The cell cycle of MTS was dominated by G0 and G1 stage.

MTS的细胞周期以G0~G1期占优势。

The G1 stage of the Hep G-2 cells exposed to ELF was delayed and the percentage of apoptotic cells was increased with the increasing of exposure time.

使Hep G-2细胞G1期阻滞，凋亡率随照射时间延长逐渐增高。

The result demonstrated that, contrast to the control, 82.5% cells were at G0/ G1 stage that were induced by serum deprivation, the induced effectiveness is significantly higher than control group ( 61.2%)( p < 0.05).

流式细胞仪检测显示，与对照组（61.2%）相比，血清饥饿能使82.5%的牛胎儿成纤维细胞进入G0/G1期，诱导效率明显高于对照组。