NMDA

Methods: Three injured models induced by caffeine, KCI, and NMDA, respectively, were used to assay the action of tanshinone in cultured primary cortex neurone of baby rats.

方法：采用组织培养法，以原代大鼠大脑皮层神经细胞为材料，制备咖啡因、KCI及NMDA诱导的钙超载损伤模型。

Results: shored The neural cytosolic Ca++ in bilirubin precipitated brain tissue was significantly more than that in the control group; NMDA receptor antagonist GAPA could significantly decrease the cytosolic Ca++ overload.

结果显示：胆红素毒性脑组织细胞内Ca2+较对照组明显升高，NMDA受体拮抗剂GAPA可显著减轻胆红素毒性脑组织细胞内Ca2+超载。

Glutamate receptors of the N-methyl-D-aspartate ( NMDA) subtype are highly expressed in the central nervous system and are involved in excitatory synaptic transmission and synaptic plasticity.

NMDA（N-甲基-D-天氡氨酸）受体是离子型谷氨酸受体的一种亚型，在中枢神经系统的突触传递和突触可塑性调节中起着重要的作用。

NMDA receptors are a subtype of excitatory transmitters-glutamate receptor in the central nervous system.

N-甲基-D-天冬氨酸受体（NMDA受体）是中枢神经系统中兴奋性递质谷氨酸受体的一种类型，属于离子型受体。

Two classes of ion-channel type glutamate receptors involve in NMDA receptors and AMPA/ KA receptors.

离子通道型谷氨酸受体包括AMPA/KA和NMDA受体两大类。

A model and simulation research of NMDA-depended bidirectional synaptic plasticity

NMDA依赖的突触长时程增强和长时程抑制模型与仿真研究

Effects of pentylenetetrazol at sub convulsant and convulsant dose on NMDA receptor subunits in rats

惊厥和亚惊厥剂量戊四唑对大鼠脑内NMDA受体亚单位蛋白表达的影响

Objective To explore the effect of NMDA receptor antagonist glutamyl amino methyl phosphonic acid ( GAPA) on the bilirubin neurotoxicity.

目的探讨兴奋性氨基酸受体拮抗剂磷酸甲基谷氨酰氨酸（GAPA）对胆红素神经毒性的影响。

N-methyl-D-aspartate ( NMDA) receptors, which belong to the ionotropic glutamate receptors, play major roles in synaptic transmission and plasticity, as well as excitotoxicity.

NMDA受体属于离子型谷氨酸受体，它在突触传递，突触可塑性及兴奋性毒性中都扮演着重要的角色。

Endogenous hydrogen sulfide ( H2S) can increase the production of cAMP, potentiate the exciting postsynaptic potential mediated by NMDA receptor and enhance the induction of hippocampal long-term potentiation.

内源性硫化氢（H2S）可以刺激神经细胞cAMP水平增加，提高NMDA受体介导的突触后兴奋性电位，提高诱导海马长时程增强。

Conclusion Loss of tail-flick reflex induced by Dyn was mediated, in part, by NMDA receptor.

结论Dyn致大鼠甩尾反射消失作用至少部分是由NMDA受体介导的。

The results above show NMDA receptor appears to be involved in the increase of excitability and the development of windup phenomenon in the spinal cord dorsal horn associated with carrageenan induced inflammation.

结果提示：角叉菜胶致炎导致脊髓背角神经元兴奋性升高和Windup；NMDA受体参与炎症痛和Windup形成。

Therefore, cytosolic Ca++ overload was found in the bilirubin precipitated brain tissue. NMDA receptor antagonist GAPA could prevent the cytosolic Ca++ overload in bilirubin-induced brain tissue.

因此：胆红素神经毒性脑组织细胞内Ca2+超载，NMDA受体拮抗剂GAPA可拮抗胆红素毒性脑组织细胞内Ca2+超载。

Methods: Use in situ hybridization and patch clamp techniques.

方法: 采用原位杂交和膜片钳观察NMDA受体的数量和功能.

Study on effect of ginsenosides on NMDA receptor of hippocampus in rat by means of autoradiography

用放射自显影术研究人参总皂甙对大鼠海马NMDA受体的影响

NMDA ( N-Methyl-D-Aspartate) receptor is a kind of ionotropic receptors, which have been widely localized in peripheral somatic tissues and visceral pain pathways.

NMDA（N甲酰D天门冬氨酸）受体是一种离子型谷氨酸受体，广泛存在于外周躯体或内脏的痛觉传导通路上。

Hypoglossal responses to superior laryngeal ( sl) and pulmonary stretch receptor ( psr) inputs effects of NMDA and non-NMDA agonists on the respiratory rhythmical discharge of the hypoglossal nerve in the medullary slice from neonatal rats

舌咽神经对喉上神经及肺牵张感受器传入冲动的反应NMDA与非NMDA受体激动剂对新生大鼠延髓脑片呼吸节律性放电的作用

A mass of research indicate that NMDA receptor have been involved in the process of synaptic plasticity.

大量研究表明，NMDA受体参与了突触可塑性过程。

Relationship between amnestic effect of enflurane or isoflurane and NMDA receptor

恩氟烷、异氟烷的遗忘作用与NMDA受体的关系

If reduced NMDA receptor activity prompts schizophrenia's symptoms, what then causes this reduction?

如果说NMDA受体活性的下降促成了精神分裂的症状, 那麽又是什麽原因造成了这种活性下降?

Objective To investigate the distribution rule of NMDA receptor protein localizated on neuron membrane surface.

目的探讨神经元膜NMDA受体蛋白分子在神经元膜表面的分布规律,对单个膜NMDA受体 蛋白进行定位.

NMDA receptors are abundantly expressed in central nervous system. It mediates the induction of synaptic plasticity, modulates gene expression, and participates in the induction of neuronal networks during various developmental stages.

NMDA受体广泛表达于中枢神经系统，介导突触可塑性的形成，调控神经元的基因表达，参与发育过程中神经网络的形成。

Effects of Dyn are mediated by both Kappa and NMDA receptor.

Dyn的病理作用是通过Kappa阿片受体和EAA的NMDA受体两种途径介导的。

Furthermore, at temperatures more close to physiological brain temperatures , more NMDA receptor mediated responses room temperature.

在重复刺激或近于生理性温度时, NMDA受体传递的反应更为明显.

Results: After hypobaric hypoxia the number of NMDA mRNA positive neurons was decreased, the open probability of NMDA receptor was reduced, the open time constant was decreased, the close time constant was increased.

结果：胎鼠低压低氧后，NMDA受体数量和通道开放机率减少，通道开放时间常数减少，通道关闭时间常数增加。

N-methyl-D-aspartate receptors ( NMDA receptors, NMDARs), as the ion channel-type glutamate receptors, are vital for mammal neuron system.

NMDA受体（N-methyl-D-aspartate receptors，NMDA受体）是哺乳动物神经系统中重要的离子通道型谷氨酸受体。

The Relationship between Surface Expression and Distribution of NMDA Receptor and Synaptogenesis and Synaptic Function in Cultured Neurons Using GFP-Tagged NMDA Receptor Subunits

活细胞可视化研究NMDA受体在神经元的表面表达、分布及其与突触形成和功能的关系

Conclusion Anoxia contributes exteriorly to the over-activation of NMDA receptors and thus radical inward current of Ca2+.

结论缺氧是使NMDA受体通道过度激活和Ca2+大量内流的一外因条件。

Entirely different mechanisms may end up explaining why NMDA receptor transmission becomes attenuated.

但是真正可以盖棺论定的研究尚未出现;到头来,很可能出现完全不同的机制,来解释为什麽NMDA受体的讯息传递遭到减弱.

The present study was to investigate whether peripheral NMDA receptors were involved in the persistent nociception induced by subcutaneous ( s.c.) bee venom injection in the conscious rat by using quatitative pain scoring methods. s.

为了探讨外周NMDA受体是否介导大鼠足底皮下注入蜜蜂毒诱导的持续伤害性行为反应，本研究应用动物痛行为学定量方法评价局部用药对持续伤害性行为反应的作用效果。