alveolar wall

The results showed epinephrine could increase the permeability of pulmonary microvascular wall and alveolar wall; there were edema, bleeding, transparent membranes in the tissue of damaged lung.

结果表明，肾上腺素可使肺血管壁、肺泡壁通透性增高，肺间质、肺泡、支气管水肿，出血及透明膜形成；

The important mechanical factors of lung injury might be tensile strain or stress in alveolar wall, and pulmonary superficial impact on the inside wall of lung.

肺损伤的重要力学因素可能是肺泡壁的拉伸应力或应变和肺表面的碰撞。

Three months after Irradiation, acute inflammation performance disappear, a large number of fibroblast proliferation and the specific performance of mesenchymal cells increased, alveolar wall thickening, fibroblast cells increased, reducing the number of blood vessels.

照射3个月后急性炎症表现消失，出现成纤维细胞的大量增生，具体表现为间质细胞数增多、肺泡壁增厚、成纤维细胞数增多、血管数减少。

SP-C immunofluorescence staining was performed to identify the type II alveolar cells in the TUNEL positive cells. Results: Destruction of alveolar wall and loss of the alveolar unit were observed in papain only group and papain+ irradiation group.

TUNEL与SP-C免疫荧光法鉴定凋亡细胞中的II型肺泡上皮细胞。结果：木瓜蛋白酶组、木瓜蛋白酶和钴60处理组大鼠出现明显肺气肿改变。

The pathological changes in lungs caused by PQ were mainly shown as congestion, leukocyte infiltration, thickness and local hemorrhage in alveolar wall.

肺组织病理学检查可见PQ中毒后肺泡壁充血、增厚，炎症细胞浸润，并有局灶性出血；

In senior group, WT mice showed significant alveolar expansion and interstitial lung damage, by contrast, u-PA gene deletion had more complete alveolar wall structure and no significant increase of alveolar space.

相比之下，u-PA基因缺失小鼠肺泡壁结构较完整，肺泡腔无明显增大。

Microscope shows pulmono-stroma fibrous tissue hyperplasia, lymphocytes and neutrophil infiltrate, alveolar wall edema and thickening, alveolus cava contain with protein-material and a bit dust cells.

光镜下肺间质纤维组织增多、淋巴及中性粒细胞浸润，肺泡壁水肿、增厚，肺泡腔内含有蛋白样物质及少许尘埃细胞。

Imaging Diagnosis and Classification of Alveolar Wall Vanishing Lung Syndrome with a Repot of 15 Cases

16例肺泡壁消失肺综合征影像学诊断及分型探讨

After oral administration, BPs significantly improved pathological injury. Inflammation were reduced, alveolar hemorrhage were extenuate, alveolar wall and inflammatory cell infiltration were decreased.

口服给药后，柴胡明显改善了病理损伤，病理切片显示给药后炎症程度明显减轻，肺泡腔出血明显减少，肺泡壁变薄，炎性细胞浸润减少。

Results Histopathological evaluation: Typical pathological inflammation changes in the lung were observed in LPS groups, including alveolar congestion, hemorrhage, edema, infiltration of neutrophils in the airspace or vessel wall, thickness of the alveolar wall, and hyaline membrane formation.

【结果】肺组织病理学评价：LPS各组大鼠肺组织呈现典型的炎症病理变化，包括肺泡充血、出血、水肿、肺泡腔和血管壁中性粒细胞浸润、肺泡壁增厚、透明膜形成等病变。

Results: The congestion and bleeding of alveolar wall were lessened after given EDTA.

结果：给予EDTA后，肺泡壁充血、出血均有减轻，且高剂量组的各时期肺泡上皮细胞增生明显，易见核分裂。

Group C alveolar wall thickening, between pulmonary edema, increased collagen deposition.

C组肺泡壁增厚，肺间质水肿，胶原纤维沉积增多。

Blood capillary of alveolar wall engorgement and pulmonary hyaline membrane are formed, and the infiltration of inflammatory cells in alveolar spaces. In the treatment group, these changes were less obvious, and the amplitude decreased.

肺泡壁毛细血管充血扩张，有肺透明膜形成。肺间质、肺泡腔内有大量炎性细胞浸润，治疗组以上损伤程度明显减轻。

But there were obvious differences in the model group, such as narrowed alveolar space, thickened alveolar wall, lung congestion, pulmonary edema, infiltration of inflammatory cells, markedly increased of cavity tablets alveolar cells, and local emphysema.

模型组肺泡腔变窄，肺泡壁增厚；肺充血，肺间质水肿，可见炎细胞浸润，肺泡腔内可见粒细胞明显增多，并见局部肺气肿。

It has also been proved in the pathological section of the animal pulmonary tissues that in comparison between Chinese and western medicine group and the model control group, the pulmonary alveoli were slightly enlarged, and alveolar wall was thickened, but the bronchial mucosa was comparatively complete.

动物肺组织病理切片证实，中西药组与模型对照组相比，肺泡轻度扩大，虽有肺泡壁增厚，但支气管黏膜较完整。

These studies demonstrated that, at least in animal models, apoptosis of alveolar wall or endothelial cells is sufficient to cause pulmonary emphysema, even without the accumulation of inflammatory cells.

动物实验证明即使没有炎性细胞的聚集，肺泡壁细胞或内皮细胞凋亡也足够引起肺气肿的发生。

Ultrastructural characteristics of adventitial myofibroblast and cell phenotype in alveolar wall vessels were observed by electron microscopy.

透射电镜观察外膜成肌纤维细胞和腺泡内血管壁细胞表型。

Light microscope ( HE): PQ group shows that focal pulmonary hemorrhage, alveolar wall thickening apparent, alveolar and interstitial pulmonary edema, inflammatory cell infiltration, and some sections can be seen focal atelectasis, emphysema.

光镜检查：PQ组可见肺灶性出血，肺泡壁明显增厚，肺泡和肺间质水肿，大量炎性细胞浸润，部分切片可见灶状肺不张，肺气肿。

Orcein staining showed that in MVAD group compared with control group there were sparser distributed and weakly stained elastic fibers in airway wall and alveolar wall, while in vascular wall the thinner ones had focal architecture distortion and fiber fracture. 4.

地衣红染色显示：MVAD组较对照组肺泡气管壁弹性纤维分布稀疏，染色浅；血管壁弹性纤维纤细，局部紊乱、断裂。

Alveolar wall congestion, alveolar infiltration and gas bubbles in pulmonary capillary were observed in lung.

红细胞压积（Hct）升高。（2）快速减压可造成兔肺泡壁充血、肺泡腔内渗出，肺毛细血管内气泡形成。

Alcohol group can be seen varying degrees of alveolar and alveolar septal infiltration of inflammatory cells, some alveolar wall damaged or collapsed.

酒精组可见不同程度的肺泡及肺泡间隔炎性细胞浸润，部分肺泡壁破坏、塌陷，间隔增宽，胶原沉积。

Results: ( 1) There were lots of infiltrating inflammation cells in the bronchial mucus and lung tissue, the destruction of the alveolar wall was observed, it was accorded with the COPD pathologic change.

结果：（1）吸烟大鼠的支气管粘膜和肺实质均有大量炎细胞浸润，肺泡壁结构破坏，形成肺大泡，符合COPD的病理改变。