deoxycytidine

Objective To investigate the expression of deoxycytidine kinase ( DCK) and cytidine deaminase ( CDA) gene in children with acute leukemia and to explore the relationship between gene expression and clinical response to cytarabine ( Ara-C).

目的通过测定急性白血病（AL）患儿脱氧胞苷激酶（DCK）和胞苷脱氨酶（CDA）基因的表达，探讨其与阿糖胞苷（Ara-C）耐药及临床疗效间的关系。

Effects of 5-Aza-2'-deoxycytidine on proliferation of human lung cancer cell lines

5-脱氧杂氮胞苷对人肺腺癌细胞系增殖的影响

Effect of 5-Aza-2'-deoxycytidine on human renal cell carcinoma cell line OS-RC-2

5-氮杂-2′-脱氧胞苷对OS-RC-2肾癌细胞生物学行为的影响

Inhibitory effect of 5-Aza-2'-deoxycytidine on proliferation of Raji cell line

甲基转移酶抑制剂对Raji细胞系增殖抑制的实验研究

Expression of Deoxycytidine Kinase and Cytidine Deaminase mRNA in Children with Acute Leukemia

急性白血病患儿脱氧胞苷激酶和胞苷脱氨酶基因表达

The effect of combination of 5-Aza-2 ′-deoxycytidine with Paclitaxel on gastric cancer cells

5-氮杂-2′-脱氧胞苷联合紫杉醇对中分化胃腺癌细胞株SGC-7901的作用

Methods To investigate the changes of DNA methyltransferase 1 ( DNMT_1) mRNA and methylation status of progesterone receptor double promotors ( PRA, PRB) before and after 5-Aza-2 ′-deoxycytidine treatment.

方法用5-杂氮-2′-脱氧胞苷处理白血病细胞株HL-60、K562和Jukat，分析处理前后DNA甲基转移酶1mRNA表达和孕酮受体双启动子（PRA、PRB）甲基化状态的变化。

Mechanism of 5-Aza-2 ′-deoxycytidine Inhibiting the Growth of CNE Cell Line

5-杂氮-2′-脱氧胞苷抑制人鼻咽癌CNE细胞生长的机制

BACKGROUND& OBJECTIVE: 5-Aza-2 ′-deoxycytidine ( 5-Aza-CdR) is an inhibitor of DNA methyltransferase; it may reactivate methylated antioncogene, therefore, inhibit the growth of cancer cells.

背景与目的：5-杂氮-2′-脱氧胞苷是DNA甲基转移酶抑制剂，能使因甲基化而失活的抑癌基因重新激活，进而抑制肿瘤的生长。

Effects of 5-Aza-2'-Deoxycytidine on the Growth and Apoptosis of Bile Duct Cancer Cell Line

5-氮-2′-脱氧胞苷对胆管癌细胞株生长周期及凋亡的影响

Objective To understand the relationship between the clinical efficacy and intracellular levels of deoxycytidine kinase ( dCK) and cytidine deaminase ( CDA) in acute leukemia.

目的了解急性白血病细胞内脱氧胞苷激酶（dCK）、胞苷脱氨酶（CDA）的含量与临床疗效的关系。

Nullification assay was used to examine whether EGCG nullify the rescue effect of deoxycytidine ( dCdR) to AraC.

头孢哌酮含量测定用流动相的改进以对消实验研究EGCG能否逆转脱氧胞苷（dCdR）的补救作用；