it is suggested that lv hypertrophy induced by pressure overload may not affect the curvilinearity of end-systolic pressure-volume relation and the lv contractility in basal state is enhanced while its responsiveness to iso is reduced in rhr.
结果提示,压力超负荷心肌肥大并不影响PIPVR的非线性特征,基础状态下,RHR肥大左室的收缩能力增强,但对异丙肾上腺素的反应性降低。
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