macrophages

Result: GLB 7 increased the production of cAMP in a concentration and time dependent manner in murine peritoneal macrophages.

结果：GLB7能剂量依赖性引起小鼠腹腔MФ中cAMP浓度快速升高，5min达峰值，之后缓慢下降，至30min时基本恢复原来水平。

Protection by New Compound FLZ and Ginkgo Biloba Extract ( EGb761) Against the Oxidization of Human Low Density Lipoprotein and Its Cytotoxicity to Macrophages and Vascular Endothelial Cells

新化合物FLZ和银杏叶提取物（EGb761）对人低密度脂蛋白过氧化及其损伤巨噬细胞及血管内皮细胞的保护作用

Electron Microscope Observation of Effect of Thymic Factor on Mouse Peritoneal Macrophages

胸腺因子对小鼠腹腔巨噬细胞影响的电镜观察

The macrophages collect to ingest the lipid material.

巨噬细胞聚集起来吞噬脂质.

Effects of macrophages on hemopoiesis after total body irradiation in mice

小鼠6Gy全身照射后巨噬细胞对造血的抑制效应研究

HLA DR Ag was strongly expressed on activated T cells , macrophages.

巨噬细胞 、 T淋巴细胞高表达HLADR抗原 (P& lt; 005).

The results showed that the decoction increased the stability of WBC, increased the phagocytic and digestive power of depressed macrophages and enhanced the recovery of T-cell immunity from depression.

结果表明，本方剂对小鼠白细胞（WBC）有稳定作用，能增强受抑制巨噬细胞（Mφ）的吞噬能力和增进Mφ对吞噬物的消化能力，并可促进受抑制T细胞免疫功能恢复。

there were lots of active fibroblasts and macrophages around the deposit.

沉淀样物附近可见活跃的成纤维细胞和巨噬细胞分布.

the positive signals all located at cytoplasm of monocyte-macrophages system.

结果显示阳性信号多位于单核 - 巨噬细胞系统细胞胞浆内.

Objective To investigate the effect of atorvastatin on CD36 expression in THP-1 macrophages.

目的研究阿托伐他汀（atorvastatin）对THP-1巨噬细胞B类清道夫受体CD36表达的影响。

macrophages, as part of the inflammatory response, produce reactivespecies in order to attack bacteria.

巨噬细胞作为炎症反应的一部分, 可以产生活性氧以抗击细菌.

Finally methods are described for the analysis of gene expression in macrophages.

最后方法被为在巨噬细胞里的基因表示的分析描述.

Extracting, purifying and activity testing of pulmonary alveolar macrophages of mice

小鼠肺泡巨噬细胞的提取、纯化及活性检测

Microscopically, Mycobacterium avium-intracellulare infection is marked by numerous acid fast organisms growing within macrophages.

显微镜下, 可见巨噬细胞内大量鸟型分枝杆菌,已用抗酸染色标记出来.

Therefore, changing the types of the macrophages could affect host tumor immunity.

因此, 改变巨噬细胞类型可以影响机体对肿瘤的免疫应答.

We suspected that certain tumor cells produce substances that prevent macrophages from finding and destroying them.

我们猜想,某些肿瘤细胞会产生使巨噬细胞不能发现和摧毁它们的一些物质.

METHODS: The thiophosphoric acid modified oligonucleotides were transferred into rat peritoneal macrophages directly, TNF content in LPS-stimulated cell culture supernatant was determined by ELISA.

方法：直接将经硫代磷酸修饰的寡核苷酸导入大鼠腹腔巨噬细胞，以减少细胞分泌产生肿瘤坏死因子（TNF）的效果作为判断指标，以ELISA法检测细胞培养上清液中的TNF-α含量。

Result : GLB 7 increased the production of IP 3 and DAG in murine peritoneal macrophages.

结果显示GLB7能引起小鼠MΦ中IP3和DAG浓度升高.

Contrasted with control and pretreatment groups, the activity and IL-2 secretion of T Lymphocytes cells and the activity of macrophages in cavitas abdominis of radio-frequency ablation ( RFA) treatment group were improved significantly ( P < 0.05).

RFA治疗组的实验兔T淋巴细胞活性、T淋巴细胞分泌IL-2的能力和腹腔巨噬细胞的活性均较手术治疗组和自身治疗前组有显著提高（P＜0.05）。

clusters of lipid-laden macrophages are seen.

可见到成簇的充满脂质的巨噬细胞.

All of these entities lack prominent foamy macrophages.

这些病变均缺少明显的泡沫细胞.

Additional pink epithelioid macrophages compose most of the rest of the granuloma.

更多的上皮样细胞构成了肉芽肿的其它部分.

These suggest that dexamethasone inhibition of CSE expression is through different mechanisms in primary macrophages and macrophage cell line and the mechanism of action of glucocorticoid on CSE expression might be related to cell types.

提示糖皮质激素可能通过不同的机制抑制原代巨噬细胞和巨噬细胞系的CSE表达，GCs对CSE表达的调节机制可能具有细胞特异性。

Experimental Study for Thrombocytopenic Purpura Therapy by Targeting Macrophages in Liver and Spleen

靶向肝脾巨噬细胞治疗血小板减少性紫癜的实验研究

Method To take sciatic nerves in 3-4 days SD rats and purify Schwann cells. A group: DMEM/ F12 contained 10% calf blood serum. B group: conditioned medium of macrophages activated by self-neural homogenate.

方法取3-4dSD乳鼠坐骨神经，纯化培养许旺细胞，A组加入含10%胎牛血清的DMEM/F12培养基，B组加入自体神经匀浆激活的巨噬细胞条件培养基；

Influence of B-17 oestradiol on peritoneal macrophages in mice

β-17雌二醇对小鼠腹腔巨噬细胞的影响

The exudate consists of edema fluid, protein, Pneumocystis organisms, and dead macrophages.

渗出物包含水肿液 、 蛋白质 、 肺孢子虫以及坏死的巨噬细胞.

The innate resistance thought to be related to the activity of macrophages.

先天性抵抗力被认为与巨噬细胞的活性有关。

Conclusions Viable M. tuberculosis H_ 37 R_v can induce the activation of macrophages and the production of more NO and cytokines which play important roles in the host immune response.

结论活的结核分支杆菌H37Rv能显著诱导巨噬细胞产生NO和细胞因子，而产生有利于宿主的免疫应答。

Cytotoxicity levels of pulmonary alveolar macrophages in experimental silicotic rats ( 0.8-1.4%) were lower than those of the control rats ( 3.3-4.0%), but after administration of Tilorone, cytotoxicity levels were raised to as high as 7.8-11.2%.

实验性矽肺大鼠肺巨噬细胞分泌的细胞毒因子活性（0.8-1.4%）比正常对照组（3.3-4.0%）为低，但给予梯络龙后，其活性高达7.8-11.2%。