reperfusion

The destruction of BBB is an important mechanism of cerebral ischemia-reperfusion injury.

血脑屏障破坏是脑缺血再灌注(ischemiareperfusion,IR) 脑损伤的重要的病理生理基础.

Assessing the Myocardium After Attempted Reperfusion: Should We Bother?

试图再灌注后评估心肌: 我们应否打扰?

The limb ischemia reperfusion injury is the hot point in clinical study especially in pathological change.

肢体缺血再灌注损伤是临床上研究热点，特别是病理改变的研究。

Effects of Shuang Shen Tong Guan ( SSTG) on NF-κ B Signal Pathway During Myocardial Ischemia-Reperfusion Injury

双参通冠方对急性心肌缺血再灌注NF-κB信号途径的影响

The effects of Gammahydroxybutyrate ( GHB) on myocardial ischemia reperfusion injury were studied in isolated Langendorff perfused rat heart preparation.

对20只SD大鼠离体Langendorff心脏，行全心缺血30min再灌注30min的方法，研究羟丁酸钠对心肌缺血再灌注损伤的作用和机制。

In reperfusion group, the thrombus was thinner and unclear.

溶栓组可见血栓模糊不清, 形态较血栓组稀薄.

Protective effects of delayed preconditioning against cell apoptosis caused by myocardial ischemia reperfusion

缺血预适应与针灸缺血延迟预适应对心肌缺血再灌注所致细胞凋亡的保护

Clinical observation of intestinal ischemia reperfusion injury in intussusception

小肠缺血再灌注损伤在肠套叠患儿的临床观察

ASI can lower expression of NF-kB and decrease cerebral ischemia-reperfusion injury.

刺五加注射液可下调的NF-kB表达,减轻脑缺血再灌注损伤.

Effect of hyperbaric oxygen on membrane function in rat retina after ischemia reperfusion.

高压氧对缺血再灌注条件下鼠视网膜细胞膜功能的影响（论著）

Pathway and Mechanism of Nitric Oxide Production in Vascular Tissue on Limb Ischemia Reperfusion Injury

肢体缺血再灌注损伤时血管组织一氧化氮产生途径及其机制

Complement Deposition and Interference by Defibrase in Cerebral Ischemia Reperfusion

鼠脑缺血再灌损伤中补体作用的探讨

Objective : To identify the relationship of hypovolemic shock and reperfusion injury.

目的: 确定创伤所致的低血容量性休克病人预后与再灌注损伤的关系.

Methods A model of retinal ischemia reperfusion in rat was made.

方法制备大鼠视网膜缺血再灌注模型.

Protective effects of verapamil and energy compounds on the ischemia reperfusion injury of animal intestine

异搏定、能量合剂对小肠缺血再灌注损伤的保护作用

Methods: The animals achieved hypercapnic acidosis with mechanical hypoventilation before ischemia-reperfusion.

方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.

Unchecked, oxygen radicals may cause reperfusion injuries and other problems.

看来, 氧基能导致多次灌注的伤害和其他问题.

Thrombolysis therapy and percutaneous transluminal coronary angioplasty recover myocardial reperfusion efficiently.

溶栓治疗和经皮冠状动脉腔内成形术有效恢复缺血心肌的再灌注.

Apoptosis is an important pathophysiological characteristic during hepatic ischemia-reperfusion injury ( HIRI ) .

细胞凋亡是肝缺血 — 再 灌注 损伤重要的病理生理特征.

Protective function of astragalus pretreatment on the myocardial ultrastructure in rats with ischemia reperfusion

黄芪预处理对缺血再灌注大鼠心肌超微结构的保护作用

Conclusion: F 2 can antagonize the arrhythmias induced by ischemia-reperfusion in rat heart.

结论: F2对大鼠心脏缺血再灌注心律失常具有拮抗作用.

Conclusions Hyperbaric oxygen therapy has good protective effect on liver ischemic reperfusion injury, and the earlier the HBO is used, the better the protective effect.

结论HBO对肝I/R具有保护作用，处理越早作用越明显。

Effects of renal ischemia reperfusion injuries on glomerular ultrastructure and anionic sites in model rats

肾缺血再灌注对大鼠肾小球超微结构及负电荷位点的影响

Conclusion The ICAM-1 would be monitoring marker of Inflammatory reaction after spinal cord ischemia/ reperfusion injury.

结论ICAM1可做为脊髓IR后炎症反应程度的监测指标。

Objective To study the level of nitric NOS in ischemia-reperfusion rat liver.

目的探讨大鼠缺血再灌注肝一氧化氮合酶的变化及其意义.

In the later reperfusion period, less H 2 O 2 generated. Intra-cellular calcium increased continuously.

再灌流晚期H2O2产生有所减少,而细胞内钙持续增高.

Neutrophil dynamic changes in ischemia reperfusion flap

大鼠岛状皮瓣缺血-再灌注损伤的中性粒细胞动态变化

ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.

ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.

Objective: To observe the pulmonary apoptosis resulted from ischemia reperfusion lung injury in rats.

目的：观察肺缺血再灌注损伤对肺细胞凋亡的影响。

The IPC 、 PMA protected the myocardium from sustained ischemia-reperfusion injury . Bre.

Bre. 则可消除IPC对心肌的保护作用.